Total submissions: 5
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV000189787 | SCV000243435 | uncertain significance | not provided | 2024-06-26 | criteria provided, single submitter | clinical testing | In silico analysis supports that this missense variant does not alter protein structure/function; Has not been previously published as pathogenic or benign to our knowledge |
Labcorp Genetics |
RCV000189787 | SCV000816776 | likely benign | not provided | 2024-12-22 | criteria provided, single submitter | clinical testing | |
Fulgent Genetics, |
RCV000764997 | SCV000896180 | uncertain significance | Neuronal ceroid lipofuscinosis 2; Autosomal recessive spinocerebellar ataxia 7 | 2018-10-31 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV002381645 | SCV002694252 | uncertain significance | Inborn genetic diseases | 2018-12-03 | criteria provided, single submitter | clinical testing | The p.T427M variant (also known as c.1280C>T), located in coding exon 11 of the TPP1 gene, results from a C to T substitution at nucleotide position 1280. The threonine at codon 427 is replaced by methionine, an amino acid with similar properties. This amino acid position is poorly conserved in available vertebrate species. In addition, this alteration is predicted to be tolerated by in silico analysis. Since supporting evidence is limited at this time, the clinical significance of this alteration remains unclear. |
Centre de Biologie Pathologie Génétique, |
RCV001252368 | SCV001428123 | uncertain significance | Intellectual disability | 2019-01-01 | no assertion criteria provided | clinical testing |