Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Counsyl | RCV000670223 | SCV000795053 | likely pathogenic | Neuronal ceroid lipofuscinosis 2 | 2017-10-25 | criteria provided, single submitter | clinical testing | |
Invitae | RCV001385878 | SCV001585886 | pathogenic | not provided | 2023-09-11 | criteria provided, single submitter | clinical testing | ClinVar contains an entry for this variant (Variation ID: 554563). This sequence change creates a premature translational stop signal (p.Leu120Serfs*18) in the TPP1 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in TPP1 are known to be pathogenic (PMID: 10330339). This variant is not present in population databases (gnomAD no frequency). This premature translational stop signal has been observed in individual(s) with neuronal ceroid lipofuscinosis (PMID: 11339651). This variant is also known as 1-bp insertion of "T". For these reasons, this variant has been classified as Pathogenic. |
Fulgent Genetics, |
RCV002493105 | SCV002801476 | pathogenic | Neuronal ceroid lipofuscinosis 2; Autosomal recessive spinocerebellar ataxia 7 | 2022-03-09 | criteria provided, single submitter | clinical testing | |
Natera, |
RCV000670223 | SCV002094875 | pathogenic | Neuronal ceroid lipofuscinosis 2 | 2021-02-17 | no assertion criteria provided | clinical testing |