Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001065547 | SCV001230508 | pathogenic | not provided | 2024-02-23 | criteria provided, single submitter | clinical testing | This sequence change affects an acceptor splice site in intron 2 of the CTSK gene. RNA analysis indicates that disruption of this splice site induces altered splicing and likely results in a shortened protein product. This variant is present in population databases (rs587663163, gnomAD 0.003%). Disruption of this splice site has been observed in individual(s) with pycnodysostosis (PMID: 10878663). In at least one individual the data is consistent with being in trans (on the opposite chromosome) from a pathogenic variant. It has also been observed to segregate with disease in related individuals. ClinVar contains an entry for this variant (Variation ID: 859435). Studies have shown that disruption of this splice site results in skipping of exon 3, but is expected to preserve the integrity of the reading-frame (PMID: 10878663). For these reasons, this variant has been classified as Pathogenic. |
Baylor Genetics | RCV003467829 | SCV004215242 | pathogenic | Pyknodysostosis | 2023-09-18 | criteria provided, single submitter | clinical testing | |
Fulgent Genetics, |
RCV003467829 | SCV005671336 | likely pathogenic | Pyknodysostosis | 2024-03-31 | criteria provided, single submitter | clinical testing |