Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Counsyl | RCV000410588 | SCV000485907 | likely pathogenic | Pyknodysostosis | 2016-02-26 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV001861371 | SCV002230180 | pathogenic | not provided | 2023-09-08 | criteria provided, single submitter | clinical testing | This sequence change affects an acceptor splice site in intron 2 of the CTSK gene. RNA analysis indicates that disruption of this splice site induces altered splicing and likely results in a shortened protein product. This variant is not present in population databases (gnomAD no frequency). Disruption of this splice site has been observed in individual(s) with pycnodysostosis (PMID: 10878663). In at least one individual the data is consistent with being in trans (on the opposite chromosome) from a pathogenic variant. It has also been observed to segregate with disease in related individuals. ClinVar contains an entry for this variant (Variation ID: 370558). Studies have shown that disruption of this splice site results in skipping of exon 3, but is expected to preserve the integrity of the reading-frame (PMID: 10878663). For these reasons, this variant has been classified as Pathogenic. |
Genome- |
RCV000410588 | SCV004049736 | likely pathogenic | Pyknodysostosis | 2023-04-11 | criteria provided, single submitter | clinical testing |