Total submissions: 1
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Clin |
RCV001225266 | SCV001397531 | pathogenic | Glanzmann thrombasthenia | 2020-09-06 | reviewed by expert panel | curation | The NM_000419.5:c.1618C>T variant results in premature termination of translation at Gln540 in exon 17/30. The resulting transcript is predicted to undergo NMD. The variant is absent from gnomAD v2.1.1 and v3 (PM2 threshold of <0.0001). The variant is reported in one compound heterozygous GT patient along with the splice site variant, c.1440-13_1440-1del (PMID: 25728920). In summary, based on the evidence available at this time, the variant is classified as pathogenic. GT-specific criteria applied: PVS1, PM2_Supporting, PM3_Supporting, PP4_Strong. |