Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Eurofins Ntd Llc |
RCV000287266 | SCV000342114 | uncertain significance | not provided | 2016-05-27 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV001239611 | SCV001412495 | pathogenic | LAMA2-related muscular dystrophy | 2023-04-06 | criteria provided, single submitter | clinical testing | For these reasons, this variant has been classified as Pathogenic. Advanced modeling of protein sequence and biophysical properties (such as structural, functional, and spatial information, amino acid conservation, physicochemical variation, residue mobility, and thermodynamic stability) has been performed at Invitae for this missense variant, however the output from this modeling did not meet the statistical confidence thresholds required to predict the impact of this variant on LAMA2 protein function. ClinVar contains an entry for this variant (Variation ID: 14297). This missense change has been observed in individuals with congenital muscular dystrophy (PMID: 30827497, 32266982; Invitae). This variant is not present in population databases (gnomAD no frequency). This sequence change replaces cysteine, which is neutral and slightly polar, with arginine, which is basic and polar, at codon 862 of the LAMA2 protein (p.Cys862Arg). |
Revvity Omics, |
RCV000287266 | SCV003815834 | uncertain significance | not provided | 2021-03-24 | criteria provided, single submitter | clinical testing | |
OMIM | RCV000015367 | SCV000035628 | pathogenic | Congenital muscular dystrophy due to partial LAMA2 deficiency | 2003-02-01 | no assertion criteria provided | literature only |