Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Women's Health and Genetics/Laboratory Corporation of America, |
RCV004782002 | SCV005395125 | uncertain significance | not specified | 2024-09-10 | criteria provided, single submitter | clinical testing | Variant summary: MAT1A c.1006G>A (p.Gly336Arg) results in a non-conservative amino acid change located in the S-adenosylmethionine synthetase, C-terminal domain (IPR022630) of the encoded protein sequence. Five of five in-silico tools predict a damaging effect of the variant on protein function. The variant allele was found at a frequency of 8e-06 in 251470 control chromosomes (gnomAD). c.1006G>A has been reported in the literature in at least an individual affected with hypermethioninemia (example: Chamberlin_2000). These data do not allow any conclusion about variant significance. In vitro functional studies show reduced activity for the variant (Chamberlin_2000). The following publication has been ascertained in the context of this evaluation (PMID: 10677294). ClinVar contains an entry for this variant (Variation ID: 1211). Based on the evidence outlined above, the variant was classified as VUS-possibly pathogenic. |
Genomic Medicine Center of Excellence, |
RCV004820815 | SCV005442308 | likely pathogenic | not provided | 2024-12-19 | criteria provided, single submitter | clinical testing | |
OMIM | RCV000001270 | SCV000021420 | pathogenic | Hepatic methionine adenosyltransferase deficiency | 2000-02-01 | no assertion criteria provided | literature only |