Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
All of Us Research Program, |
RCV003996096 | SCV004837565 | uncertain significance | Hypertrophic cardiomyopathy | 2023-10-06 | criteria provided, single submitter | clinical testing | This missense variant replaces proline with alanine at codon 95 of the MYL2 protein. Computational prediction is inconclusive regarding the impact of this variant on protein structure and function (internally defined REVEL score threshold 0.5 < inconclusive < 0.7, PMID: 27666373). Functional analysis of purified human proteins expressing this variant has shown a decrease in calcium binding affinity (PMID: 11102452); the clinical relevance of this observation is not known. This variant has been reported in an individual affected with hypertrophic cardiomyopathy (PMID: 8673105). This variant has not been identified in the general population by the Genome Aggregation Database (gnomAD). The available evidence is insufficient to determine the role of this variant in disease conclusively. Therefore, this variant is classified as a Variant of Uncertain Significance. |
OMIM | RCV000015110 | SCV000035367 | pathogenic | Hypertrophic cardiomyopathy 10 | 1996-05-01 | no assertion criteria provided | literature only | |
Leiden Muscular Dystrophy |
RCV000015110 | SCV000045758 | not provided | Hypertrophic cardiomyopathy 10 | 2012-03-26 | no assertion provided | curation |