Total submissions: 2
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Institute Of Molecular Biology And Genetics, |
RCV000513098 | SCV000607740 | likely pathogenic | Familial isolated restrictive cardiomyopathy | 2017-05-15 | criteria provided, single submitter | research | The patient presented with typical signs of restrictive cardiomyopathy at 24 y.o. No signs of system disorder haven been noted. The family history did not report a consanguinity with homozygous status of the mutation suspect with condition. |
Labcorp Genetics |
RCV000639673 | SCV000761253 | uncertain significance | Hypertrophic cardiomyopathy 10 | 2022-08-31 | criteria provided, single submitter | clinical testing | In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. Algorithms developed to predict the effect of sequence changes on RNA splicing suggest that this variant may disrupt the consensus splice site. This sequence change affects the initiator methionine of the MYL2 mRNA. The next in-frame methionine is located at codon 20. This variant is not present in population databases (gnomAD no frequency). Disruption of the initiator codon has been observed in individual(s) with hypertrophic cardiomyopathy (PMID: 30297972). ClinVar contains an entry for this variant (Variation ID: 444868). |