Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV000494542 | SCV000582904 | pathogenic | not provided | 2019-08-27 | criteria provided, single submitter | clinical testing | In-frame deletion of 3 amino acids in a non-repeat region; Not observed at a significant frequency in large population cohorts (Lek et al., 2016); In silico analysis, which includes protein predictors and evolutionary conservation, supports a deleterious effect; This variant is associated with the following publications: (PMID: 18625437, 20167518, 10598813, 10498624, 32040803) |
Labcorp Genetics |
RCV000002333 | SCV000956351 | pathogenic | Granulomatous disease, chronic, autosomal recessive, cytochrome b-positive, type 2 | 2024-01-26 | criteria provided, single submitter | clinical testing | This variant, c.55_63del, results in the deletion of 3 amino acid(s) of the NCF2 protein (p.Lys19_Asp21del), but otherwise preserves the integrity of the reading frame. This variant is present in population databases (rs796065033, gnomAD 0.003%). This variant has been observed in individual(s) with chronic granulomatous disease (PMID: 10498624, 10598813, 18625437; Invitae). In at least one individual the data is consistent with being in trans (on the opposite chromosome) from a pathogenic variant. ClinVar contains an entry for this variant (Variation ID: 2245). For these reasons, this variant has been classified as Pathogenic. |
OMIM | RCV000002333 | SCV000022491 | pathogenic | Granulomatous disease, chronic, autosomal recessive, cytochrome b-positive, type 2 | 1999-10-01 | no assertion criteria provided | literature only |