Total submissions: 5
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Eurofins Ntd Llc |
RCV000395782 | SCV000345640 | uncertain significance | not provided | 2018-02-01 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV002519349 | SCV003268661 | uncertain significance | Zellweger spectrum disorders | 2022-09-15 | criteria provided, single submitter | clinical testing | This sequence change replaces proline, which is neutral and non-polar, with leucine, which is neutral and non-polar, at codon 674 of the PEX1 protein (p.Pro674Leu). This variant is present in population databases (rs141509344, gnomAD 0.01%). This missense change has been observed in individual(s) with PEX1-related conditions (PMID: 32203225). ClinVar contains an entry for this variant (Variation ID: 290970). Advanced modeling of protein sequence and biophysical properties (such as structural, functional, and spatial information, amino acid conservation, physicochemical variation, residue mobility, and thermodynamic stability) performed at Invitae indicates that this missense variant is not expected to disrupt PEX1 protein function. In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. |
Ambry Genetics | RCV002519350 | SCV003706190 | uncertain significance | Inborn genetic diseases | 2021-09-15 | criteria provided, single submitter | clinical testing | (Thomas, 2020) Based on insufficient or conflicting evidence, the clinical significance of this alteration remains unclear. |
Department of Pathology and Laboratory Medicine, |
RCV005396905 | SCV006058054 | uncertain significance | Peroxisome biogenesis disorder 1A (Zellweger); Heimler syndrome 1; Peroxisome biogenesis disorder 1B | 2023-09-21 | criteria provided, single submitter | research | |
Prevention |
RCV004752836 | SCV005353336 | uncertain significance | PEX1-related disorder | 2024-09-04 | no assertion criteria provided | clinical testing | The PEX1 c.2021C>T variant is predicted to result in the amino acid substitution p.Pro674Leu. This variant was reported in an individual with Zellweger syndrome, who also had two pathogenic variants in PEX1 (Thomas et al. 2020. PubMed ID: 32203225). This variant is reported in 0.0099% of alleles in individuals of Ashkenazi Jewish descent in gnomAD. At this time, the clinical significance of this variant is uncertain due to the absence of conclusive functional and genetic evidence. |