Total submissions: 5
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
CFTR2 | RCV000577122 | SCV000924243 | pathogenic | Cystic fibrosis | 2017-12-08 | reviewed by expert panel | research | |
CFTR- |
RCV001009535 | SCV001169630 | pathogenic | Cystic fibrosis; CFTR-related disorder | 2018-03-09 | criteria provided, single submitter | curation | the variant causes a phenotype but regarding our data, we can't formally attribute it to CF, CFTR-RD or both |
Labcorp Genetics |
RCV000577122 | SCV001576101 | likely pathogenic | Cystic fibrosis | 2024-02-22 | criteria provided, single submitter | clinical testing | This sequence change replaces proline, which is neutral and non-polar, with leucine, which is neutral and non-polar, at codon 99 of the CFTR protein (p.Pro99Leu). This variant is present in population databases (rs397508467, gnomAD 0.002%). This missense change has been observed in individuals with cystic fibrosis (PMID: 11732487, 14872121). ClinVar contains an entry for this variant (Variation ID: 53610). Advanced modeling of protein sequence and biophysical properties (such as structural, functional, and spatial information, amino acid conservation, physicochemical variation, residue mobility, and thermodynamic stability) performed at Invitae indicates that this missense variant is expected to disrupt CFTR protein function with a positive predictive value of 80%. Experimental studies have shown that this missense change affects CFTR function (PMID: 8663008, 29805046). In summary, the currently available evidence indicates that the variant is pathogenic, but additional data are needed to prove that conclusively. Therefore, this variant has been classified as Likely Pathogenic. |
Genetics and Genomic Medicine Centre, |
RCV000577122 | SCV005873748 | pathogenic | Cystic fibrosis | 2021-05-29 | criteria provided, single submitter | clinical testing | |
Clin |
RCV000577122 | SCV000679420 | not provided | Cystic fibrosis | no assertion provided | literature only |