Total submissions: 6
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV000227990 | SCV000285007 | likely benign | Cystic fibrosis | 2024-03-13 | criteria provided, single submitter | clinical testing | |
Quest Diagnostics Nichols Institute San Juan Capistrano | RCV000506184 | SCV000601120 | uncertain significance | not specified | 2016-12-05 | criteria provided, single submitter | clinical testing | |
CFTR- |
RCV001009482 | SCV001169577 | pathogenic | CFTR-related disorder | 2018-03-26 | criteria provided, single submitter | curation | |
Mayo Clinic Laboratories, |
RCV001508590 | SCV001714847 | uncertain significance | not provided | 2019-12-12 | criteria provided, single submitter | clinical testing | |
Johns Hopkins Genomics, |
RCV000227990 | SCV004024521 | uncertain significance | Cystic fibrosis | 2023-06-06 | criteria provided, single submitter | clinical testing | This CFTR intronic variant (rs878854021) is rare (<0.1%) in a large population dataset (gnomADv3.1.2: 6/152142 total alleles; 0.004%; no homozygotes) and has been reported in ClinVar (Variation ID:237857). It has not been reported in the literature in individuals with cystic fibrosis, to our knowledge. Bioinformatic analysis predicts that this intronic variant may weaken the native acceptor splice site although this has not been confirmed experimentally to our knowledge. We consider the clinical significance of CFTR c.4243-7del to be uncertain at this time. |
ARUP Laboratories, |
RCV001508590 | SCV004564331 | uncertain significance | not provided | 2023-02-07 | criteria provided, single submitter | clinical testing | The CFTR c.4243-7del variant (rs878854021), to our knowledge, is not reported in the medical literature but is reported in the CFTR- France database in 2 individuals with CBAVD when found in trans with another pathogenic variant (see link). This variant is reported in ClinVar (Variation ID: 237857) and is found in the non-Finnish European population with an allele frequency of 0.005% (5/112352 alleles) in the Genome Aggregation Database. This is an intronic deletion, and computational analyses (Alamut Visual Plus v.1.5.1) predict that this variant may impact splicing by weakening the nearby canonical acceptor splice site. Due to limited information, the clinical significance of this variant is uncertain at this time. References: Link to CFTR France: https://cftr.iurc.montp.inserm.fr/cftr/ |