Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Ce |
RCV001263432 | SCV001441469 | likely benign | Hereditary angioedema type 3 | criteria provided, single submitter | clinical testing | The c.41T>C (p.Leu14Ser) variant, located in exon 1 of the F12 gene, was identified in a single type I C1-INH-HAE Bulgarian patient co-carrying a pathogenic frameshift mutation in SERPING1 gene. Bioinformatic analysis by SIFT and PolyPhen2 algorithms predicted deleterious and probably damaging, respectively. It has been detected in 0.01556% alleles worldwide (gnomAD database) and its allele frequency is greater than that expected for FXII-HAE. Taking all the above into account and according to ACMG Guidelines (Criteria: BS1, BP5) the variant is considered likely benign. | |
Labcorp Genetics |
RCV002069380 | SCV002416933 | likely benign | not provided | 2024-01-22 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV004035415 | SCV004864911 | uncertain significance | Inborn genetic diseases | 2023-11-03 | criteria provided, single submitter | clinical testing | The c.41T>C (p.L14S) alteration is located in exon 1 (coding exon 1) of the F12 gene. This alteration results from a T to C substitution at nucleotide position 41, causing the leucine (L) at amino acid position 14 to be replaced by a serine (S). Based on insufficient or conflicting evidence, the clinical significance of this alteration remains unclear. |