Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Women's Health and Genetics/Laboratory Corporation of America, |
RCV000586108 | SCV000697166 | likely pathogenic | Tay-Sachs disease | 2016-06-09 | criteria provided, single submitter | clinical testing | Variant summary: The HEXA c.187G>T (p.Glu63X) variant results in a premature termination codon, predicted to cause a truncated or absent HEXA protein due to nonsense mediated decay, which are commonly known mechanisms for disease. The variant of interest was not observed in controls (ExAC, 1000 Gs or ESP), nor has it been, to our knowledge, reported in affected individuals via publications and/or reputable clinical laboratories/databases. Therefore, taking all available lines of evidence into consideration, the variant of interest has been classified as a "Probable Disease Variant/Likely Pathogenic," until additional information becomes available. |
Labcorp Genetics |
RCV000586108 | SCV003450471 | pathogenic | Tay-Sachs disease | 2024-03-05 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Glu63*) in the HEXA gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in HEXA are known to be pathogenic (PMID: 1833974, 8490625). This variant is present in population databases (no rsID available, gnomAD 0.003%). This variant has not been reported in the literature in individuals affected with HEXA-related conditions. ClinVar contains an entry for this variant (Variation ID: 496010). For these reasons, this variant has been classified as Pathogenic. |
Counsyl | RCV000586108 | SCV000792481 | likely pathogenic | Tay-Sachs disease | 2017-06-26 | no assertion criteria provided | clinical testing | |
Natera, |
RCV000586108 | SCV002089760 | likely pathogenic | Tay-Sachs disease | 2017-08-17 | no assertion criteria provided | clinical testing |