Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Color Diagnostics, |
RCV001189122 | SCV001356333 | uncertain significance | Familial hypercholesterolemia | 2023-04-26 | criteria provided, single submitter | clinical testing | This variant is located in the intron 6 splice donor region of the LDLR gene. Computational splicing tools and conservation analyses are inconclusive regarding the impact of this variant on RNA splicing. To our knowledge, functional assays have not been performed for this variant nor has this variant been reported in individuals affected with familial hypercholesterolemia in the literature. This variant has been identified in 2/250868 chromosomes in the general population by the Genome Aggregation Database (gnomAD). Available evidence is insufficient to determine the role of this variant in disease conclusively. Therefore, this variant is classified as a Variant of Uncertain Significance. |
All of Us Research Program, |
RCV004010339 | SCV004827422 | uncertain significance | Hypercholesterolemia, familial, 1 | 2023-05-16 | criteria provided, single submitter | clinical testing | This variant is located in the intron 6 splice donor region of the LDLR gene. Computational splicing tools and conservation analyses are inconclusive regarding the impact of this variant on RNA splicing. To our knowledge, functional assays have not been performed for this variant nor has this variant been reported in individuals affected with familial hypercholesterolemia in the literature. This variant has been identified in 2/250868 chromosomes in the general population by the Genome Aggregation Database (gnomAD). Available evidence is insufficient to determine the role of this variant in disease conclusively. Therefore, this variant is classified as a Variant of Uncertain Significance. |
Ambry Genetics | RCV004639491 | SCV005136011 | uncertain significance | Cardiovascular phenotype | 2024-05-17 | criteria provided, single submitter | clinical testing | The c.940+4A>C intronic variant results from an A to C substitution 4 nucleotides after coding exon 6 in the LDLR gene. This nucleotide position is well conserved in available vertebrate species. In silico splice site analysis for this alteration is inconclusive. Based on the available evidence, the clinical significance of this variant remains unclear. |