Total submissions: 3
| Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
|---|---|---|---|---|---|---|---|---|
| Labcorp Genetics |
RCV001053594 | SCV001217864 | pathogenic | Charcot-Marie-Tooth disease, type I | 2021-12-24 | criteria provided, single submitter | clinical testing | This variant is also known as p.Gly138Arg. ClinVar contains an entry for this variant (Variation ID: 208149). Algorithms developed to predict the effect of missense changes on protein structure and function are either unavailable or do not agree on the potential impact of this missense change (SIFT: "Deleterious"; PolyPhen-2: "Possibly Damaging"; Align-GVGD: "Class C0"). Experimental studies have shown that this missense change affects MPZ function (PMID: 29687021). Algorithms developed to predict the effect of sequence changes on RNA splicing suggest that this variant may create or strengthen a splice site. For these reasons, this variant has been classified as Pathogenic. This missense change has been observed in individual(s) with Dejerine-Sottas disease (PMID: 7506095, 10399750, 12242557). In at least one individual the variant was observed to be de novo. This sequence change replaces glycine, which is neutral and non-polar, with arginine, which is basic and polar, at codon 167 of the MPZ protein (p.Gly167Arg). This variant is not present in population databases (gnomAD no frequency). |
| Gene |
RCV000194294 | SCV000243906 | not provided | Charcot-Marie-Tooth disease type 1B | no assertion provided | literature only | ||
| Inherited Neuropathy Consortium | RCV000789484 | SCV000928840 | uncertain significance | Charcot-Marie-Tooth disease | no assertion criteria provided | literature only |