Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV000550782 | SCV000644532 | pathogenic | Tuberous sclerosis 2 | 2024-06-16 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Arg1451Alafs*25) in the TSC2 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in TSC2 are known to be pathogenic (PMID: 10205261, 17304050). The frequency data for this variant in the population databases is considered unreliable, as metrics indicate poor data quality at this position in the gnomAD database. This premature translational stop signal has been observed in individual(s) with tuberous sclerosis (PMID: 21520333). ClinVar contains an entry for this variant (Variation ID: 64920). For these reasons, this variant has been classified as Pathogenic. |
Baylor Genetics | RCV003460650 | SCV004205114 | pathogenic | Isolated focal cortical dysplasia type II | 2022-11-21 | criteria provided, single submitter | clinical testing | |
Gene |
RCV005401315 | SCV006060127 | likely pathogenic | not provided | 2024-10-22 | criteria provided, single submitter | clinical testing | Frameshift variant predicted to result in protein truncation or nonsense mediated decay in a gene for which loss of function is a known mechanism of disease; This variant is associated with the following publications: (PMID: 32647919, 35918040, 31031587) |
Tuberous sclerosis database |
RCV000055122 | SCV000083340 | not provided | Tuberous sclerosis syndrome | no assertion provided | curation |