Total submissions: 5
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Revvity Omics, |
RCV001780680 | SCV002022030 | likely pathogenic | Maple syrup urine disease | 2023-04-12 | criteria provided, single submitter | clinical testing | |
Fulgent Genetics, |
RCV004571095 | SCV002809922 | likely pathogenic | Maple syrup urine disease type 1A | 2024-02-24 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV001780680 | SCV003523014 | pathogenic | Maple syrup urine disease | 2024-01-31 | criteria provided, single submitter | clinical testing | This sequence change replaces alanine, which is neutral and non-polar, with valine, which is neutral and non-polar, at codon 216 of the BCKDHA protein (p.Ala216Val). This variant is present in population databases (rs369448982, gnomAD 0.01%). This missense change has been observed in individual(s) with maple syrup urine disease and/or MSUD (PMID: 16786533, 31980395, 34556729). In at least one individual the data is consistent with being in trans (on the opposite chromosome) from a pathogenic variant. ClinVar contains an entry for this variant (Variation ID: 1323975). An algorithm developed to predict the effect of missense changes on protein structure and function (PolyPhen-2) suggests that this variant is likely to be tolerated. For these reasons, this variant has been classified as Pathogenic. |
Baylor Genetics | RCV004571095 | SCV004215867 | likely pathogenic | Maple syrup urine disease type 1A | 2024-03-04 | criteria provided, single submitter | clinical testing | |
Prevention |
RCV003892844 | SCV004711572 | pathogenic | BCKDHA-related disorder | 2024-01-30 | no assertion criteria provided | clinical testing | The BCKDHA c.647C>T variant is predicted to result in the amino acid substitution p.Ala216Val. This variant has been reported in patients with autosomal recessive maple syrup urine disease (Rodriguez-Pombo et al. 2006. PubMed ID: 16786533; Table S1, Strauss et al. 2020. PubMed ID: 31980395; Fang et al. 2021. PubMed ID: 34556729). This variant is reported in 0.013% of alleles in individuals of African descent in gnomAD. This variant is interpreted as pathogenic. |