Total submissions: 2
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Molecular Genetics Laboratory, |
RCV001199478 | SCV001162521 | pathogenic | Retinitis pigmentosa | 2020-01-09 | criteria provided, single submitter | research | |
Labcorp Genetics |
RCV001306605 | SCV001495985 | likely pathogenic | not provided | 2023-04-24 | criteria provided, single submitter | clinical testing | This variant is not present in population databases (gnomAD no frequency). In summary, the currently available evidence indicates that the variant is pathogenic, but additional data are needed to prove that conclusively. Therefore, this variant has been classified as Likely Pathogenic. ClinVar contains an entry for this variant (Variation ID: 813047). This variant has not been observed in the literature in individuals with autosomal recessive IMPDH1-related conditions. This variant has been reported in individual(s) with autosomal dominant retinitis pigmentosa (PMID: 32531858); however, the role of the variant in this condition is currently unclear. This sequence change affects an acceptor splice site in intron 3 of the IMPDH1 gene. It is expected to disrupt RNA splicing. Variants that disrupt the donor or acceptor splice site typically lead to a loss of protein function (PMID: 16199547), and loss-of-function variants in IMPDH1 are known to be pathogenic (PMID: 14981049, 33090715). |