Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Institute of Medical Genetics and Applied Genomics, |
RCV001268848 | SCV001448053 | pathogenic | not provided | 2020-10-23 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV004659451 | SCV005155372 | pathogenic | Cardiovascular phenotype | 2024-05-14 | criteria provided, single submitter | clinical testing | The c.2423dupA pathogenic mutation, located in coding exon 12 of the PKP2 gene, results from a duplication of A at nucleotide position 2423, causing a translational frameshift with a predicted alternate stop codon (p.N809Efs*18). This variant has been detected in individuals with features consistent with arrhythmogenic right ventricular cardiomyopathy (Biernacka EK et al. J Appl Genet, 2021 Dec;62:613-620; Ambry internal data). This variant is considered to be rare based on population cohorts in the Genome Aggregation Database (gnomAD). This alteration is expected to result in loss of function by premature protein truncation or nonsense-mediated mRNA decay. As such, this alteration is interpreted as a disease-causing mutation. |
Mayo Clinic Laboratories, |
RCV001268848 | SCV005414066 | pathogenic | not provided | 2024-08-02 | criteria provided, single submitter | clinical testing | PM2, PS4_supporting, PVS1 |