Total submissions: 6
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Laboratory for Molecular Medicine, |
RCV000038210 | SCV000061878 | likely benign | not specified | 2012-05-22 | criteria provided, single submitter | clinical testing | Asp829Asp in exon 12 of PKP2: This variant is not expected to have clinical sign ificance because it does not alter an amino acid residue and is not located with in the splice consensus sequence. It has been identified in 2/7020 European Amer ican chromosomes and 2/3738 African American chromosomes from a broad population by the NHLBI Exome Sequencing Project (http://evs.gs.washington.edu/EVS; dbSNP rs142362933). Asp829Asp in exon 12 of PKP2 (rs142362933; allele frequency = 2/3 738) ** |
Labcorp Genetics |
RCV000462231 | SCV000557313 | likely benign | Arrhythmogenic right ventricular dysplasia 9 | 2024-01-22 | criteria provided, single submitter | clinical testing | |
Color Diagnostics, |
RCV000777804 | SCV000913799 | likely benign | Cardiomyopathy | 2018-10-18 | criteria provided, single submitter | clinical testing | |
Gene |
RCV001650871 | SCV001863241 | benign | not provided | 2015-03-03 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV002426566 | SCV002741769 | likely benign | Cardiovascular phenotype | 2019-05-02 | criteria provided, single submitter | clinical testing | This alteration is classified as likely benign based on a combination of the following: seen in unaffected individuals, population frequency, intact protein function, lack of segregation with disease, co-occurrence, RNA analysis, in silico models, amino acid conservation, lack of disease association in case-control studies, and/or the mechanism of disease or impacted region is inconsistent with a known cause of pathogenicity. |
Breakthrough Genomics, |
RCV001650871 | SCV005216607 | likely benign | not provided | criteria provided, single submitter | not provided |