Total submissions: 2
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001956267 | SCV002240076 | pathogenic | Charcot-Marie-Tooth disease axonal type 2P | 2021-01-16 | criteria provided, single submitter | clinical testing | This variant has not been reported in the literature in individuals with LRSAM1-related conditions. This sequence change creates a premature translational stop signal (p.Gln697*) in the LRSAM1 gene. While this is not anticipated to result in nonsense mediated decay, it is expected to disrupt the last 27 amino acid(s) of the LRSAM1 protein. This variant is not present in population databases (ExAC no frequency). This variant disrupts the region of the LRSAM1 protein between p.Cys694Arg and p.Leu708Argfs*28. This region has been determined to be associated with autosomal dominant LRSAM1-related conditions (PMID: 27615052, 27164712, 22012984), which suggests that variants that occur in this region are likely to be clinically significant. For these reasons, this variant has been classified as Pathogenic. |
Genome |
RCV001956267 | SCV004228846 | not provided | Charcot-Marie-Tooth disease axonal type 2P | no assertion provided | phenotyping only | Variant interpreted as Pathogenic and reported on 01-28-2021 by Lab Invitae. GenomeConnect-Invitae Patient Insights Network assertions are reported exactly as they appear on the patient-provided report from the testing laboratory. Registry team members make no attempt to reinterpret the clinical significance of the variant. Phenotypic details are available under supporting information. |