Total submissions: 2
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Center For Human Genetics And Laboratory Diagnostics, |
RCV001726706 | SCV001961028 | pathogenic | Polycystic kidney disease, adult type | 2021-04-09 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV004039992 | SCV005006684 | pathogenic | Inborn genetic diseases | 2024-01-08 | criteria provided, single submitter | clinical testing | The c.10538dupA (p.R3514Efs*112) alteration, located in exon 35 (coding exon 35) of the PKD1 gene, consists of a duplication of A at position 10538, causing a translational frameshift with a predicted alternate stop codon after 112 amino acids. This alteration is expected to result in loss of function by premature protein truncation or nonsense-mediated mRNA decay. This variant was not reported in population-based cohorts in the Genome Aggregation Database (gnomAD). Based on the available evidence, this alteration is classified as pathogenic. |