Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV001765459 | SCV001998151 | uncertain significance | not provided | 2024-06-03 | criteria provided, single submitter | clinical testing | Not observed at significant frequency in large population cohorts (gnomAD); In silico analysis supports that this missense variant has a deleterious effect on protein structure/function; Has not been previously published as pathogenic or benign to our knowledge |
Fulgent Genetics, |
RCV002506774 | SCV002816127 | likely pathogenic | Polycystic kidney disease, adult type | 2024-02-22 | criteria provided, single submitter | clinical testing | |
Clinical Genomics Laboratory, |
RCV003458176 | SCV004177145 | uncertain significance | Autosomal dominant polycystic kidney disease | 2023-07-28 | criteria provided, single submitter | clinical testing | The PKD1 c.9598G>T (p.Val3200Phe) variant, to our knowledge, has not been reported in the medical literature. This variant has been reported in the ClinVar database as a variant of uncertain significance by two submitters (ClinVar ID 1309290). The PKD1 c.9598G>T (p.Val3200Phe) variant is absent from the general population (gnomAD v.2.1.1), indicating it is not a common variant. Computational predictors are uncertain as to the impact of this variant on PKD1 function. Due to limited information, and based on ACMG/AMP guidelines for variant interpretation (Richards S et al., PMID: 25741868), the clinical significance of this variant is uncertain. |