Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Institute for Genomic Medicine |
RCV001249692 | SCV001423688 | pathogenic | Developmental and epileptic encephalopathy, 4 | 2018-05-16 | criteria provided, single submitter | clinical testing | [ACMG/AMP: PVS1, PS2, PM2] This alteration is a null variant in a gene where LOF is a known mechanism of disease [PVS1], is de novo in origin as it was not detected in the submitted parental specimens (identity confirmed) [PS2], is absent from or rarely observed in large-scale population databases [PM2]. |
Labcorp Genetics |
RCV003770290 | SCV004642455 | pathogenic | Early infantile epileptic encephalopathy with suppression bursts | 2023-11-02 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Arg464Glnfs*31) in the STXBP1 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in STXBP1 are known to be pathogenic (PMID: 20887364, 26384463). This variant is not present in population databases (gnomAD no frequency). This premature translational stop signal has been observed in individual(s) with clinical features of STXBP1-related conditions (PMID: 32371413, 33176815). ClinVar contains an entry for this variant (Variation ID: 973268). For these reasons, this variant has been classified as Pathogenic. |
Genome |
RCV001265422 | SCV001443548 | pathogenic | Infantile epilepsy syndrome | 2019-02-04 | no assertion criteria provided | provider interpretation | Submission from Simons Searchlight facilitated by GenomeConnect. Variant interpreted by the Simons Searchlight team most recently on 2019-02-04 and interpreted as Pathogenic. Variant was initially reported on 2018-05-16 by GTR ID of laboratory name 196472. The reporting laboratory might also submit to ClinVar. |