Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Revvity Omics, |
RCV001780917 | SCV002024028 | likely pathogenic | not provided | 2021-10-07 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV002034585 | SCV002238687 | pathogenic | Severe combined immunodeficiency due to DCLRE1C deficiency | 2023-09-03 | criteria provided, single submitter | clinical testing | This variant is present in population databases (rs767758218, gnomAD 0.006%). This sequence change creates a premature translational stop signal (p.Ile543Argfs*12) in the DCLRE1C gene. While this is not anticipated to result in nonsense mediated decay, it is expected to disrupt the last 150 amino acid(s) of the DCLRE1C protein. For these reasons, this variant has been classified as Pathogenic. This variant disrupts a region of the DCLRE1C protein in which other variant(s) (p.Thr557Asnfs*21) have been determined to be pathogenic (PMID: 26476407). This suggests that this is a clinically significant region of the protein, and that variants that disrupt it are likely to be disease-causing. ClinVar contains an entry for this variant (Variation ID: 1324212). This variant has not been reported in the literature in individuals affected with DCLRE1C-related conditions. |
Baylor Genetics | RCV004571097 | SCV005059359 | likely pathogenic | Histiocytic medullary reticulosis | 2024-02-09 | criteria provided, single submitter | clinical testing |