Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
DASA | RCV002298438 | SCV002588792 | pathogenic | Bronchiectasis with or without elevated sweat chloride 2 | 2022-11-03 | criteria provided, single submitter | clinical testing | The c.1522C>T;p.(Arg508*) variant creates a premature translational stop signal in the SCNN1A gene. It is expected to result in an absent or disrupted protein product - PVS1. This sequence change has been observed in affected individual(s) and ClinVar contains an entry for this variant (Clinvar ID:9264; PMID: 10510337) - PS4. The variant is present at low allele frequencies population databases (rs137852634 – gnomAD 0.0001768%; ABraOM no frequency - https://abraom.ib.usp.br/) - PM2_supporting. In summary, the currently available evidence indicates that the variant is pathogenic. |
Fulgent Genetics, |
RCV002482850 | SCV002790246 | likely pathogenic | Pseudohypoaldosteronism, type IB1, autosomal recessive; Bronchiectasis with or without elevated sweat chloride 2; Liddle syndrome 3 | 2022-02-16 | criteria provided, single submitter | clinical testing | |
Genomic Medicine Center of Excellence, |
RCV002298438 | SCV005442103 | uncertain significance | Bronchiectasis with or without elevated sweat chloride 2 | 2024-12-19 | criteria provided, single submitter | clinical testing | |
OMIM | RCV000009846 | SCV000030067 | pathogenic | Pseudohypoaldosteronism, type IB1, autosomal recessive | 1999-10-01 | no assertion criteria provided | literature only |