Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001483366 | SCV001687756 | likely benign | not provided | 2024-12-16 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV004037238 | SCV004946813 | uncertain significance | Inborn genetic diseases | 2023-12-19 | criteria provided, single submitter | clinical testing | The c.3772A>G (p.T1258A) alteration is located in exon 32 (coding exon 31) of the SI gene. This alteration results from a A to G substitution at nucleotide position 3772, causing the threonine (T) at amino acid position 1258 to be replaced by an alanine (A). Based on insufficient or conflicting evidence, the clinical significance of this alteration remains unclear. |
Prevention |
RCV003946263 | SCV004759187 | uncertain significance | SI-related disorder | 2023-12-11 | no assertion criteria provided | clinical testing | The SI c.3772A>G variant is predicted to result in the amino acid substitution p.Thr1258Ala. To our knowledge, this variant has not been reported in the literature. This variant is reported in 0.18% of alleles in individuals of African descent in gnomAD, which may be too high to be causative of disease. Although we suspect that this variant may be benign, at this time, the clinical significance of this variant is uncertain due to the absence of conclusive functional and genetic evidence. |