Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001885190 | SCV002237110 | pathogenic | not provided | 2024-12-12 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Tyr1417*) in the SI gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in SI are known to be pathogenic (PMID: 16329100, 23103650, 25452324). This variant is present in population databases (rs142090504, gnomAD 0.01%). This premature translational stop signal has been observed in individual(s) with irritable bowel syndrome (PMID: 29408290). ClinVar contains an entry for this variant (Variation ID: 1325070). For these reasons, this variant has been classified as Pathogenic. |
Gene |
RCV001885190 | SCV002562614 | uncertain significance | not provided | 2023-07-24 | criteria provided, single submitter | clinical testing | Reported in an individuals with irritable bowel syndrome, although this variant was also present in controls (Garcia-Etxebarria et al., 2018; Zheng et al., 2021); Nonsense variant predicted to result in protein truncation or nonsense mediated decay in a gene for which loss-of-function is a known mechanism of disease; This variant is associated with the following publications: (PMID: 34186061, 32732636, 29408290, 33375084) |
Fulgent Genetics, |
RCV001783747 | SCV002810433 | likely pathogenic | Sucrase-isomaltase deficiency | 2023-12-21 | criteria provided, single submitter | clinical testing |