Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Blueprint Genetics | RCV001074167 | SCV001239737 | likely pathogenic | Retinal dystrophy | 2019-02-19 | criteria provided, single submitter | clinical testing | |
Ocular Genomics Institute, |
RCV001376248 | SCV001573322 | likely pathogenic | Neuronal ceroid lipofuscinosis 3 | 2021-04-08 | criteria provided, single submitter | research | The CLN3 c.751C>T variant was identified in an individual with retinitis pigmentosa with a presumed recessive inheritance pattern. Through a review of available evidence we were able to apply the following criteria: PVS1, PM2. Based on this evidence we have classified this variant as Likely Pathogenic. |
Labcorp Genetics |
RCV001862820 | SCV002231827 | pathogenic | Neuronal ceroid lipofuscinosis | 2023-08-18 | criteria provided, single submitter | clinical testing | For these reasons, this variant has been classified as Pathogenic. ClinVar contains an entry for this variant (Variation ID: 866297). This variant has not been reported in the literature in individuals affected with CLN3-related conditions. This variant is not present in population databases (gnomAD no frequency). This sequence change creates a premature translational stop signal (p.Gln251*) in the CLN3 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in CLN3 are known to be pathogenic (PMID: 9311735, 28542676). |
Baylor Genetics | RCV001376248 | SCV004214312 | likely pathogenic | Neuronal ceroid lipofuscinosis 3 | 2023-10-02 | criteria provided, single submitter | clinical testing |