Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
3billion | RCV001808882 | SCV002059101 | pathogenic | Cutis laxa with severe pulmonary, gastrointestinal and urinary anomalies | 2022-01-03 | criteria provided, single submitter | clinical testing | The variant has been reported to be associated with LTBP4 related disorder (PMID:31273557). Stop-gained (nonsense): predicted to result in a loss or disruption of normal protein function through nonsense-mediated decay (NMD) or protein truncation. Multiple pathogenic variants are reported downstream of the variant (PVS1_VS). It is not observed in the gnomAD v2.1.1 dataset (PM2_M). Therefore, this variant is classified as pathogenic according to the recommendation of ACMG/AMP guideline. |
Center for Genomic Medicine, |
RCV001808882 | SCV004805404 | uncertain significance | Cutis laxa with severe pulmonary, gastrointestinal and urinary anomalies | 2024-03-25 | criteria provided, single submitter | research | |
Gene |
RCV004720944 | SCV005328003 | likely pathogenic | not provided | 2024-02-29 | criteria provided, single submitter | clinical testing | Nonsense variant predicted to result in protein truncation or nonsense mediated decay in a gene for which loss of function is a known mechanism of disease; Observed with a second truncating LTBP4 variant, phase (in cis or trans) unknown, in a patient with unspecified congenital anomalies in published literature (PMID: 31273557); Not observed at significant frequency in large population cohorts (gnomAD); Also known as c.2230C>T (p.Arg744*); This variant is associated with the following publications: (PMID: 31273557) |