Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Laboratory for Molecular Medicine, |
RCV000825524 | SCV000966839 | likely pathogenic | Muscular dystrophy-dystroglycanopathy (congenital with brain and eye anomalies), type A, 4 | 2018-08-08 | criteria provided, single submitter | clinical testing | The p.His177SerfsX2 variant in FKTN has not been previously reported in individu als with muscular dystrophy, and was absent from large population studies. This variant is predicted to cause a frameshift, which alters the protein?s amino aci d sequence beginning at position 177 and leads to a premature termination codon 2 amino acids downstream. This alteration is then predicted to lead to a truncat ed or absent protein. In summary, although additional studies are required to fu lly establish its clinical significance, the p.His177SerfsX2 variant is likely p athogenic. ACMG/AMP Criteria applied: PVS1, PM2. |
Labcorp Genetics |
RCV001825682 | SCV002167648 | pathogenic | Walker-Warburg congenital muscular dystrophy | 2021-05-04 | criteria provided, single submitter | clinical testing | This variant is not present in population databases (ExAC no frequency). This variant has not been reported in the literature in individuals with FKTN-related conditions. ClinVar contains an entry for this variant (Variation ID: 666969). For these reasons, this variant has been classified as Pathogenic. This sequence change creates a premature translational stop signal (p.His177Serfs*2) in the FKTN gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in FKTN are known to be pathogenic (PMID: 17044012, 17878207, 18752264). |
Baylor Genetics | RCV004569793 | SCV005057814 | likely pathogenic | Dilated cardiomyopathy 1X | 2024-01-13 | criteria provided, single submitter | clinical testing | |
Natera, |
RCV001825682 | SCV002079578 | likely pathogenic | Walker-Warburg congenital muscular dystrophy | 2020-03-03 | no assertion criteria provided | clinical testing |