Total submissions: 2
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV002751338 | SCV003027720 | pathogenic | not provided | 2023-06-22 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Thr1244Serfs*51) in the WDR62 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in WDR62 are known to be pathogenic (PMID: 20729831). This variant is present in population databases (rs767667321, gnomAD 0.02%). This variant has not been reported in the literature in individuals affected with WDR62-related conditions. ClinVar contains an entry for this variant (Variation ID: 1981207). For these reasons, this variant has been classified as Pathogenic. |
3billion | RCV003152802 | SCV003841641 | pathogenic | Microcephaly 2, primary, autosomal recessive, with or without cortical malformations | 2023-02-23 | criteria provided, single submitter | clinical testing | The variant is observed at an extremely low frequency in the gnomAD v2.1.1 dataset (total allele frequency: 0.002%). This variant was predicted to result in a loss or disruption of normal protein function through nonsense-mediated decay (NMD) or protein truncation. Multiple pathogenic variants are reported downstream of the variant. Therefore, this variant is classified as Pathogenic according to the recommendation of ACMG/AMP guideline. |