Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001382426 | SCV001581190 | pathogenic | Dyskeratosis congenita | 2020-03-03 | criteria provided, single submitter | clinical testing | This sequence change replaces arginine with serine at codon 282 of the TINF2 protein (p.Arg282Ser). The arginine residue is highly conserved and there is a moderate physicochemical difference between arginine and serine. This variant is not present in population databases (ExAC no frequency). This variant has been observed in in individual(s) with dyskeratosis congenita (PMID: 18252230). In at least one individual the variant was observed to be de novo. ClinVar contains an entry for this variant (Variation ID: 5626). This variant has been reported to affect TINF2 protein function (PMID: 21536674). This variant disrupts the p.Arg282 amino acid residue in TINF2. Other variants that disrupt this residue have been determined to be pathogenic (PMID: 18669893, 19090550, 21199492, 26859482, 29742735, 23094712). This suggests that this residue is clinically significant, and that variants that disrupt this residue are likely to be disease-causing. For these reasons, this variant has been classified as Pathogenic. |
OMIM | RCV000005980 | SCV000026162 | pathogenic | Dyskeratosis congenita, autosomal dominant 3 | 2011-07-01 | no assertion criteria provided | literature only | |
Gene |
RCV000032168 | SCV000055747 | not provided | Dyskeratosis congenita, autosomal dominant 1 | no assertion provided | literature only |