Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Invitae | RCV001382026 | SCV001580621 | pathogenic | Fanconi anemia | 2020-04-18 | criteria provided, single submitter | clinical testing | This sequence change results in a premature translational stop signal in the FANCI gene (p.Arg1299*). While this is not anticipated to result in nonsense mediated decay, it is expected to disrupt the last 30 amino acids of the FANCI protein. This variant is not present in population databases (ExAC no frequency). This variant has been observed in individual(s) with autosomal recessive Fanconi anemia (PMID: 17452773, 17460694). This variant has been reported to affect FANCI protein function (PMID: 20971953). This variant disrupts the C-terminus of the FANCI protein. Other variant(s) that disrupt this region (p.Asp1301Glyfs*3) have been observed in individuals with FANCI-related conditions (PMID: 17460694). This suggests that this may be a clinically significant region of the protein. For these reasons, this variant has been classified as Pathogenic. |
Fulgent Genetics, |
RCV001194987 | SCV002801773 | likely pathogenic | Fanconi anemia complementation group I | 2022-03-02 | criteria provided, single submitter | clinical testing | |
Leiden Open Variation Database | RCV001194987 | SCV001364863 | pathogenic | Fanconi anemia complementation group I | 2011-02-07 | no assertion criteria provided | curation | Curator: Arleen D. Auerbach. Submitter to LOVD: Arleen D. Auerbach. |