Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001912597 | SCV002169925 | pathogenic | Fanconi anemia | 2023-06-02 | criteria provided, single submitter | clinical testing | This variant has not been reported in the literature in individuals affected with FANCI-related conditions. This variant is present in population databases (rs771312042, gnomAD no frequency). ClinVar contains an entry for this variant (Variation ID: 1397747). For these reasons, this variant has been classified as Pathogenic. This sequence change creates a premature translational stop signal (p.Gln171*) in the FANCI gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in FANCI are known to be pathogenic (PMID: 17452773, 17460694). |
Prevention |
RCV003401850 | SCV004118886 | likely pathogenic | FANCI-related disorder | 2023-06-23 | criteria provided, single submitter | clinical testing | The FANCI c.511C>T variant is predicted to result in premature protein termination (p.Gln171*). To our knowledge, this variant has not been reported in the literature. This variant is reported in a single heterozygous individual of unknown descent in gnomAD (http://gnomad.broadinstitute.org/variant/15-89806657-C-T). Nonsense variants in FANCI are expected to be pathogenic. This variant is interpreted as likely pathogenic. |
Fulgent Genetics, |
RCV005006185 | SCV005631302 | likely pathogenic | Fanconi anemia complementation group I | 2024-02-15 | criteria provided, single submitter | clinical testing |