Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Athena Diagnostics | RCV000993001 | SCV001145670 | uncertain significance | not provided | 2019-05-01 | criteria provided, single submitter | clinical testing | |
MGZ Medical Genetics Center | RCV001832309 | SCV002580032 | uncertain significance | Familial hypokalemia-hypomagnesemia | 2022-05-31 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV000993001 | SCV003443526 | likely pathogenic | not provided | 2023-08-10 | criteria provided, single submitter | clinical testing | ClinVar contains an entry for this variant (Variation ID: 805468). In summary, the currently available evidence indicates that the variant is pathogenic, but additional data are needed to prove that conclusively. Therefore, this variant has been classified as Likely Pathogenic. Advanced modeling of protein sequence and biophysical properties (such as structural, functional, and spatial information, amino acid conservation, physicochemical variation, residue mobility, and thermodynamic stability) performed at Invitae indicates that this missense variant is expected to disrupt SLC12A3 protein function. This missense change has been observed in individuals with clinical features of Gitelman syndrome (PMID: 17329572, 30596175, 31672324). This variant is present in population databases (rs778585043, gnomAD 0.004%). This sequence change replaces serine, which is neutral and polar, with leucine, which is neutral and non-polar, at codon 350 of the SLC12A3 protein (p.Ser350Leu). |
Natera, |
RCV001832309 | SCV002089342 | uncertain significance | Familial hypokalemia-hypomagnesemia | 2020-08-19 | no assertion criteria provided | clinical testing |