Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
European Hospital Georges Pompidou Genetics Department, |
RCV002245171 | SCV002513841 | likely pathogenic | Familial hypokalemia-hypomagnesemia | 2022-04-27 | criteria provided, single submitter | clinical testing | ACMG criteria used:PS3, PM2 |
Women's Health and Genetics/Laboratory Corporation of America, |
RCV004587331 | SCV005076207 | uncertain significance | not specified | 2024-04-15 | criteria provided, single submitter | clinical testing | Variant summary: SLC12A3 c.2548+255G>A is located at a position not widely known to affect splicing. Several computational tools predict a significant impact on normal splicing: Three predict the variant abolishes a cryptic 5' donor site. At least one publication reports experimental evidence that this variant affects mRNA splicing, reporting inclusion of a 90-bp pseudoexon in intron 21 detected by midigene splicing assay (e.g. Viering_2023). The frequency of this variant in the general population could not be determined as the technology used for large population databases (ExAC, gnomAD, ESP, 1000G) cannot detect variants of this type. c.2548+255G>A has been reported in the literature in at least one compound heterozygous individual affected with Gitelman syndrome (e.g. Viering_2023). This report does not provide unequivocal conclusions about association of the variant with Familial Hypokalemia-Hypomagnesemia. The following publication has been ascertained in the context of this evaluation (PMID: 36302598). ClinVar contains an entry for this variant (Variation ID: 1684172). Based on the evidence outlined above, the variant was classified as VUS-possibly pathogenic. |
Fulgent Genetics, |
RCV002245171 | SCV005640174 | likely pathogenic | Familial hypokalemia-hypomagnesemia | 2024-04-12 | criteria provided, single submitter | clinical testing | |
Prevention |
RCV003933708 | SCV004762329 | likely benign | SLC12A3-related disorder | 2019-11-12 | no assertion criteria provided | clinical testing | This variant is classified as likely benign based on ACMG/AMP sequence variant interpretation guidelines (Richards et al. 2015 PMID: 25741868, with internal and published modifications). |