Total submissions: 2
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV000657995 | SCV000779766 | pathogenic | not provided | 2024-01-22 | criteria provided, single submitter | clinical testing | Nonsense variant predicted to result in protein truncation or nonsense mediated decay in a gene for which loss of function is a known mechanism of disease; Not observed at significant frequency in large population cohorts (gnomAD); Has not been previously published as pathogenic or benign to our knowledge |
Labcorp Genetics |
RCV001382612 | SCV001581453 | pathogenic | Episodic ataxia type 2; Developmental and epileptic encephalopathy, 42 | 2020-09-06 | criteria provided, single submitter | clinical testing | Loss-of-function variants in CACNA1A are known to be pathogenic (PMID: 10371528, 19486177, 25735478, 27250579). This variant has not been reported in the literature in individuals with CACNA1A-related conditions. ClinVar contains an entry for this variant (Variation ID: 546157). This variant is not present in population databases (ExAC no frequency). This sequence change creates a premature translational stop signal (p.Gln848*) in the CACNA1A gene. It is expected to result in an absent or disrupted protein product. For these reasons, this variant has been classified as Pathogenic. |