Total submissions: 5
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV000530425 | SCV000648369 | likely benign | Dilated cardiomyopathy 1DD | 2024-11-24 | criteria provided, single submitter | clinical testing | |
Center for Advanced Laboratory Medicine, |
RCV000852424 | SCV000995108 | uncertain significance | Arrhythmogenic right ventricular cardiomyopathy | 2017-12-22 | criteria provided, single submitter | clinical testing | |
CHEO Genetics Diagnostic Laboratory, |
RCV001798892 | SCV002042302 | uncertain significance | Cardiomyopathy | 2020-02-12 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV004659102 | SCV005160749 | uncertain significance | Cardiovascular phenotype | 2024-05-03 | criteria provided, single submitter | clinical testing | The p.R791W variant (also known as c.2371C>T), located in coding exon 9 of the RBM20 gene, results from a C to T substitution at nucleotide position 2371. The arginine at codon 791 is replaced by tryptophan, an amino acid with dissimilar properties. This alteration has been reported in a dilated cardiomyopathy (DCM) cohort; however, clinical details were limited (Horvat C et al. Genet Med, 2019 Jan;21:133-143). This amino acid position is well conserved in available vertebrate species. In addition, the in silico prediction for this alteration is inconclusive. Based on the available evidence, the clinical significance of this variant remains unclear. |
Fulgent Genetics, |
RCV000530425 | SCV005664688 | uncertain significance | Dilated cardiomyopathy 1DD | 2024-05-03 | criteria provided, single submitter | clinical testing |