Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001209558 | SCV001380998 | pathogenic | Landau-Kleffner syndrome | 2023-07-18 | criteria provided, single submitter | clinical testing | This missense change has been observed in individual(s) with clinical features of GRIN2A-related conditions (PMID: 30544257). In at least one individual the variant was observed to be de novo. This sequence change replaces glycine, which is neutral and non-polar, with serine, which is neutral and polar, at codon 498 of the GRIN2A protein (p.Gly498Ser). This variant is not present in population databases (gnomAD no frequency). ClinVar contains an entry for this variant (Variation ID: 930862). Advanced modeling of protein sequence and biophysical properties (such as structural, functional, and spatial information, amino acid conservation, physicochemical variation, residue mobility, and thermodynamic stability) performed at Invitae indicates that this missense variant is expected to disrupt GRIN2A protein function. For these reasons, this variant has been classified as Pathogenic. |
Institute of Human Genetics, |
RCV001209558 | SCV002026435 | likely pathogenic | Landau-Kleffner syndrome | 2019-01-01 | criteria provided, single submitter | research | |
Centre for Mendelian Genomics, |
RCV001209558 | SCV001367461 | uncertain significance | Landau-Kleffner syndrome | 2019-04-30 | flagged submission | clinical testing | This variant was classified as: Uncertain significance. The available evidence on this variant's pathogenicity is insufficient or conflicting. The following ACMG criteria were applied in classifying this variant: PM2,PP2,PP3. |
Clinical Genetics Laboratory, |
RCV001209558 | SCV002011769 | likely pathogenic | Landau-Kleffner syndrome | 2021-08-17 | no assertion criteria provided | clinical testing |