Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001051404 | SCV001215557 | likely benign | Familial aplasia of the vermis | 2023-10-18 | criteria provided, single submitter | clinical testing | |
Fulgent Genetics, |
RCV002497403 | SCV002779574 | uncertain significance | Joubert syndrome 3 | 2022-04-08 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV004619491 | SCV005112649 | uncertain significance | Inborn genetic diseases | 2024-04-15 | criteria provided, single submitter | clinical testing | The c.1642C>T (p.R548C) alteration is located in exon 12 (coding exon 10) of the AHI1 gene. This alteration results from a C to T substitution at nucleotide position 1642, causing the arginine (R) at amino acid position 548 to be replaced by a cysteine (C). Based on insufficient or conflicting evidence, the clinical significance of this alteration remains unclear. |
Prevention |
RCV004731083 | SCV005335806 | uncertain significance | AHI1-related disorder | 2024-09-12 | no assertion criteria provided | clinical testing | The AHI1 c.1642C>T variant is predicted to result in the amino acid substitution p.Arg548Cys. To our knowledge, this variant has not been reported in the literature. This variant is reported in 0.031% of alleles in individuals of African descent in gnomAD. At this time, the clinical significance of this variant is uncertain due to the absence of conclusive functional and genetic evidence. |