Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001051209 | SCV001215352 | pathogenic | Familial aplasia of the vermis | 2024-02-19 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Arg235Lysfs*12) in the AHI1 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in AHI1 are known to be pathogenic (PMID: 15322546, 16453322, 28442542, 29186038). The frequency data for this variant in the population databases is considered unreliable, as metrics indicate poor data quality at this position in the gnomAD database. This variant has not been reported in the literature in individuals affected with AHI1-related conditions. ClinVar contains an entry for this variant (Variation ID: 847622). For these reasons, this variant has been classified as Pathogenic. |
Blueprint Genetics | RCV001074546 | SCV001240137 | likely pathogenic | Retinal dystrophy | 2018-03-13 | criteria provided, single submitter | clinical testing | |
Gene |
RCV004773263 | SCV005386275 | pathogenic | not provided | 2024-04-22 | criteria provided, single submitter | clinical testing | Frameshift variant predicted to result in nonsense mediated decay in a gene for which loss of function is a known mechanism of disease; Not observed at significant frequency in large population cohorts (gnomAD); This variant is associated with the following publications: (PMID: 31964843, 33777383) |
Fulgent Genetics, |
RCV005036340 | SCV005666357 | likely pathogenic | Joubert syndrome 3 | 2024-03-01 | criteria provided, single submitter | clinical testing |