Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Illumina Laboratory Services, |
RCV000032737 | SCV000407403 | likely pathogenic | Autosomal recessive congenital ichthyosis 2 | 2017-04-28 | criteria provided, single submitter | clinical testing | The ALOX12B c.1294C>T (p.Arg432Ter) variant is a stop-gained variant. The p.Arg432Ter variant has been reported in two studies and identified in three individuals with congenital ichthyosis in a compound heterozygous state, two of whom were related (Eckl et al. 2005; Akiyama et al. 2010). The p.Arg432Ter variant was absent from 250 ethnically-matched controls and is not found in the 1000 Genomes Project, the Exome Sequencing Project, or the Exome Aggregation Consortium. Due to the potential impact of stop-gained variants and limited evidence, the p.Arg432Ter variant is classified as likely pathogenic for congenital ichthyosis. This variant was observed by ICSL as part of a predisposition screen in an ostensibly healthy population. |
Gene |
RCV000760415 | SCV000890292 | pathogenic | not provided | 2018-11-06 | criteria provided, single submitter | clinical testing | The R432X pathogenic variant in the ALOX12B gene has been reported previously either in the homozygous state or in combination with another ALOX12B variant in several individuals with congenital ichthyosis (Eckl et al., 2005; Kurban et al., 2010; Buckova et al., 2016). This variant is predicted to cause loss of normal protein function either through protein truncation or nonsense-mediated mRNA decay. The R432X variant is not observed in large population cohorts (Lek et al., 2016). We interpret R432X as a pathogenic variant. |
OMIM | RCV000032737 | SCV000056501 | pathogenic | Autosomal recessive congenital ichthyosis 2 | 2005-10-01 | no assertion criteria provided | literature only | |
Institute for Human Genetics, |
RCV000032737 | SCV001477963 | pathogenic | Autosomal recessive congenital ichthyosis 2 | 2021-01-07 | no assertion criteria provided | clinical testing |