Total submissions: 5
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001387158 | SCV001587711 | pathogenic | not provided | 2024-12-22 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Lys1945Serfs*42) in the EYS gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in EYS are known to be pathogenic (PMID: 18836446, 20333770). This variant is present in population databases (rs774455587, gnomAD 0.05%). This premature translational stop signal has been observed in individuals with retinitis pigmentosa (PMID: 20333770, 28704921). It has also been observed to segregate with disease in related individuals. ClinVar contains an entry for this variant (Variation ID: 1074003). For these reasons, this variant has been classified as Pathogenic. |
Baylor Genetics | RCV001826173 | SCV004192913 | pathogenic | Retinitis pigmentosa 25 | 2024-03-03 | criteria provided, single submitter | clinical testing | |
Fulgent Genetics, |
RCV001826173 | SCV005670590 | pathogenic | Retinitis pigmentosa 25 | 2024-05-24 | criteria provided, single submitter | clinical testing | |
Gene |
RCV001387158 | SCV005689862 | pathogenic | not provided | 2021-09-08 | criteria provided, single submitter | clinical testing | Frameshift variant predicted to result in protein truncation or nonsense mediated decay in a gene for which loss-of-function is a known mechanism of disease; This variant is associated with the following publications: (PMID: 32728228, 32141364, 28704921, 31964843, 20333770) |
Natera, |
RCV001826173 | SCV002084340 | pathogenic | Retinitis pigmentosa 25 | 2020-08-17 | no assertion criteria provided | clinical testing |