Total submissions: 2
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV003625208 | SCV004413793 | pathogenic | X-linked myopathy with postural muscle atrophy | 2022-11-09 | criteria provided, single submitter | clinical testing | For these reasons, this variant has been classified as Pathogenic. This sequence change creates a premature translational stop signal (p.Tyr208*) in the FHL1 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in FHL1 are known to be pathogenic (PMID: 18179888, 19687455, 19716112, 22523091, 24114807). This variant is not present in population databases (gnomAD no frequency). This variant has not been reported in the literature in individuals affected with FHL1-related conditions. |
Molecular Genetics Laboratory, |
RCV003625208 | SCV005620007 | pathogenic | X-linked myopathy with postural muscle atrophy | 2025-01-08 | criteria provided, single submitter | clinical testing | null (truncating) variant in a gene where loss of function is a known mechanism of disease (PVS1), variant not present in gnomAD general population (v4.1.0) (PM2), reported as pathogenic (ClinVar Variation ID: 2813074) without independent laboratory evaluation (PP5); detected in a proband with cardiac arrest and after the successful cardiopulmonary resuscitation; ACMG PVS1, PM2, PP5 |