Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Counsyl | RCV000668686 | SCV000793328 | likely pathogenic | Nemaline myopathy 2 | 2017-08-10 | criteria provided, single submitter | clinical testing | |
Invitae | RCV000668686 | SCV001381679 | pathogenic | Nemaline myopathy 2 | 2024-01-08 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Arg6289*) in the NEB gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in NEB are known to be pathogenic (PMID: 25205138). The frequency data for this variant in the population databases is considered unreliable, as metrics indicate poor data quality at this position in the gnomAD database. This variant has not been reported in the literature in individuals affected with NEB-related conditions. ClinVar contains an entry for this variant (Variation ID: 553276). Algorithms developed to predict the effect of sequence changes on RNA splicing suggest that this variant may disrupt the consensus splice site. For these reasons, this variant has been classified as Pathogenic. |
Greenwood Genetic Center Diagnostic Laboratories, |
RCV003233799 | SCV003932181 | pathogenic | NEB-related disorder | 2023-03-07 | criteria provided, single submitter | clinical testing | PVS1, PS3_Moderate, PM2 |
Baylor Genetics | RCV003472105 | SCV004200126 | likely pathogenic | Arthrogryposis multiplex congenita 6 | 2023-09-11 | criteria provided, single submitter | clinical testing |