Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001946731 | SCV002235819 | pathogenic | Nemaline myopathy 2 | 2021-10-21 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Leu1770*) in the NEB gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in NEB are known to be pathogenic (PMID: 25205138). This variant is not present in population databases (gnomAD no frequency). This variant has not been reported in the literature in individuals affected with NEB-related conditions. For these reasons, this variant has been classified as Pathogenic. |
Myriad Genetics, |
RCV001946731 | SCV002603069 | likely pathogenic | Nemaline myopathy 2 | 2022-03-30 | criteria provided, single submitter | clinical testing | NM_001271208.1(NEB):c.5309T>A(L1770*) is expected to be pathogenic in the context of NEB-related nemaline myopathy. This variant is predicted to lead to an abnormal or absent protein product due to the creation of a premature termination codon in NEB, a gene where loss-of-function variants are known to be pathogenic. Please note: this variant was assessed in the context of healthy population screening. |
Baylor Genetics | RCV004571728 | SCV005052109 | likely pathogenic | Arthrogryposis multiplex congenita 6 | 2024-02-08 | criteria provided, single submitter | clinical testing |