Total submissions: 6
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Ce |
RCV001090363 | SCV001245870 | pathogenic | not provided | 2017-04-01 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV001857344 | SCV002245605 | pathogenic | Early infantile epileptic encephalopathy with suppression bursts | 2022-10-23 | criteria provided, single submitter | clinical testing | This missense change has been observed in individuals with familial hemiplegic migraine (PMID: 19332696, 30498473). It has also been observed to segregate with disease in related individuals. This variant is not present in population databases (gnomAD no frequency). This sequence change replaces phenylalanine, which is neutral and non-polar, with leucine, which is neutral and non-polar, at codon 1499 of the SCN1A protein (p.Phe1499Leu). ClinVar contains an entry for this variant (Variation ID: 12902). For these reasons, this variant has been classified as Pathogenic. Advanced modeling of protein sequence and biophysical properties (such as structural, functional, and spatial information, amino acid conservation, physicochemical variation, residue mobility, and thermodynamic stability) performed at Invitae indicates that this missense variant is expected to disrupt SCN1A protein function. |
Mayo Clinic Laboratories, |
RCV001090363 | SCV002520019 | pathogenic | not provided | 2021-10-07 | criteria provided, single submitter | clinical testing | PP1_strong, PP3, PP4, PM1, PM2, PS3, PS4_moderate |
OMIM | RCV000013765 | SCV000034012 | pathogenic | Migraine, familial hemiplegic, 3 | 2009-03-31 | no assertion criteria provided | literature only | |
Uni |
RCV000013765 | SCV000091043 | not provided | Migraine, familial hemiplegic, 3 | no assertion provided | not provided | ||
Channelopathy- |
RCV003992154 | SCV004809222 | not provided | Severe myoclonic epilepsy in infancy | no assertion provided | literature only |